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By Gaw, Allan; Shepherd, James

Given the ongoing improvement of latest statins and their expanding, helpful use in medical perform and given the significance of medical learn into the atherosclerotic plaque and its position in cardiovascular and endovascular ailments, given the colossal advances in animal and human experiences that experience taken position over the last few years at a progressively expanding velocity, Gaw and Shepherd have selected subject matters on lipids Read more...

summary: Given the ongoing improvement of recent statins and their expanding, important use in scientific perform and given the significance of scientific examine into the atherosclerotic plaque and its position in cardiovascular and endovascular illnesses, given the enormous advances in animal and human stories that experience taken position over the last few years at a gradually expanding velocity, Gaw and Shepherd have selected subject matters on lipids and atherosclerosis, that are of present curiosity for researchers and clinicians alike. accordingly, the necessity for a common up to date evaluation of those advances is apparent

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61. 62. 63. 64. 65. on coronary atheroma: the Multicentre Anti-Atheroma Study (MAAS) [published erratum appears in Lancet 1994; 344(8924):762], Lancet 1994; 344(8923):633–8. Pitt B, Mancini GB, Ellis SG, Rosman HS, Park JS, McGovern ME, Pravastatin limitation of atherosclerosis in the coronary arteries (PLAC I): reduction in atherosclerosis progression and clinical events. PLAC I investigation, J Am Coll Cardiol 1995; 26(5): 1133–9. Jukema JW, Bruschke AV, van Boven AJ et al, Effects of lipid lowering by pravastatin on progression and regression of coronary artery disease in symptomatic men with normal to moderately elevated serum cholesterol levels.

The Regression Growth Evaluation Statin Study (REGRESS), Circulation 1995; 91(10):2528–40. Shepherd J, Cobbe SM, Ford I et al, Prevention of coronary heart disease with pravastatin in men with hypercholesterolemia. West of Scotland Coronary Prevention Study Group, N Engl J Med 1995; 333(20):1301–7. Sacks FM, Pfeffer MA, Moye LA et al, The effect of pravastatin on coronary events after myocardial infarction in patients with average cholesterol levels. Cholesterol and Recurrent Events Trial investigators, N Engl J Med 1996; 335(14):1001–9.

32 Immunohistochemical studies have demonstrated that T-cells are present in atheromatous plaques. 33 T-cells are of the T-helper type 1 class, characterized by the production of interleukin-2 and interferon gamma (IFN-γ). These factors can alter plaque morphology, and can also upregulate adhesion molecule expression on endothelial cells, and thus recruit more cells to join the inflammatory process. It is now generally recognized that the progression and consequences of atherosclerosis are determined by dynamic interactions between inflammatory cells recruited in response to subendothelial lipid accumulation and the local reparative ‘wound-healing’ response of surrounding VSMCs.

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